- Our Doctors
- Mr Richard Stacey
Mr Richard Stacey
MBBS (London) FRCS, FRCS (surg. neurology), Neurosurgeon
Languages spoken
English
Overview
Centres
Specialises in
Mr Richard Stacey is a Consultant Neurosurgeon specialising in neuro-oncology, spinal and intracranial conditions. He qualified from the Royal Free Medical School in 1989. Richard started his neurosurgical training at Queen’s Square, followed by three years at the Walton Centre in Liverpool before returning to Queen’s Square to complete his training. In 1999 he was appointed visiting fellow and Consultant in Neurosurgery at the Sir Charles Gairdner, Hospital in Perth Western Australia.
Richard was appointed as Consultant Neurosurgeon at the John Radcliffe in January 2000. He is an Hon. Senior Clinical Lecturer at the University of Oxford. He is widely published in both peer reviewed neuroscience journals and text books.
Career positions
Current position(s)
- Consultant Neurosurgeon specialising in neuro-oncology, spinal and intracranial conditions at Oxford University NHS Trust
- Senior Clinical Lecturer at the University of Oxford
Past position(s)
- Visiting fellow and Consultant in Neurosurgery at the Sir Charles Gairdner, Hospital in Perth Western Australia
Year qualified
- 1989
Expertise and interests
Interests
- Translational research using fMRI, DTI and spectroscopy in the treatment of patients with brain tumours and epilepsy
- Brain surgery
- Neuro surgery
Professional memberships
- Royal College of Surgeons
- Edinburgh British Society of Neurological Surgeons
- British Neuro Oncology Society
Publications
- A comparison of 2-hydroxyglutarate detection at 3 and 7 T with long-TE semi-LASER.
- Resting connectivity predicts task activation in pre-surgical populations.
- Improved localisation for 2-hydroxyglutarate detection at 3T using long-TE semi-LASER.
- Primary intraosseous meningioma: an osteosclerotic bone tumour mimicking malignancy.
- Noninvasive Quantification of 2-Hydroxyglutarate in Human Gliomas with IDH1 and IDH2 Mutations.
- Thalamo-Cortical Disruption Contributes to Short-Term Memory Deficits in Patients with Medial Temporal Lobe Damage.